Linked from

The 29 pages that link to Adenosine, each with the reason it gives.

• Adenosine triphosphateRelated: ATP breakdown can yield adenosine, which has distinct signaling effects.

• SleepRelated: Caffeine promotes alertness partly by blocking adenosine receptors.

• CaffeineRelated: Caffeine blocks receptors through which adenosine promotes sleepiness.

• MethotrexateRelated: In low-dose therapy, increased extracellular adenosine contributes to methotrexate's anti-inflammatory effects.

• Sleep hygieneRelated: Caffeine blocks adenosine receptors, helping explain why late caffeine can delay sleep.

• Sleep homeostasisRelated: Its accumulation during prolonged wakefulness is one proposed source of rising sleep pressure.

• Sleep-wake cycleRelated: Its accumulation helps explain why extended wakefulness makes sleep more likely.

• Supraventricular tachycardiaRelated: Its transient AV-node block can stop AV-node-dependent SVT or clarify the rhythm.

• Xanthine oxidaseRelated: Its degradation can feed purine breakdown pathways that ultimately generate xanthine.

• Adenosine monophosphateRelated: Removing AMP’s phosphate group produces adenosine.

• Atrioventricular nodeRelated: Its temporary suppression of atrioventricular nodal conduction can terminate nodal-dependent tachycardias.

• Nicotinamide adenine dinucleotide phosphateRelated: An adenosine unit forms one end of the NADP molecule.

• MethylxanthineRelated: Methylxanthines resemble adenosine closely enough to occupy its receptors without activating them.

• SomnolenceRelated: Its wake-dependent buildup helps explain increasing somnolence over the day.

• TachycardiaRelated: It can terminate certain re-entrant supraventricular rhythms and help reveal the underlying rhythm.

• Wolff–Parkinson–White syndromeRelated: Clinicians may use it for certain stable, regular tachycardias, but rhythm type determines its safety.

• WakefulnessRelated: Its buildup helps make prolonged wakefulness harder to sustain.

• SomnologyRelated: Its buildup offers a biochemical account of the drive to sleep.