Linked from

The 75 pages that link to Apoptosis, each with the reason it gives.

• MetamorphosisRelated: Cell death removes larval structures, such as a tadpole's tail, during transformation.

• CancerRelated: Cancer cells can evade this process and survive when normal cells would die.

• Developmental biologyRelated: Programmed cell death sculpts structures and removes cells during development.

• HypoxiaRelated: Hypoxic stress can activate apoptosis, depending on its severity and duration.

• ChemotherapyRelated: Drug-induced cellular damage can trigger this route to cancer-cell death.

• Cell biologyRelated: It shows how cellular life includes controlled self-removal as well as growth.

• DNA damageRelated: Severe or irreparable damage can trigger this protective response.

• NeurodegenerationRelated: Apoptotic pathways can execute neuronal death after severe or persistent stress.

• OntogenyRelated: Programmed cell removal shapes developing tissues and continues to affect organisms later in life.

• Signal transductionRelated: Signaling networks can activate or suppress the pathways that execute cell death.

• TadpoleRelated: Programmed cell death helps remove the tail and reshape tissues during metamorphosis.

• Flow cytometryRelated: Fluorescent probes can distinguish apoptotic cells from viable and necrotic cells.

• CarcinogenesisRelated: Cancer-prone cells may evade this form of programmed cell death.

• CisplatinRelated: Persistent cisplatin-induced DNA damage can trigger this response in cancer cells.

• EmbryologyRelated: Programmed cell death removes or reshapes cells during normal development.

• OrganogenesisRelated: Localized cell removal sculpts structures such as separated digits and hollow organs.

• PupaRelated: Programmed cell death helps dismantle larval tissues that are not retained in the adult.

• OncogeneRelated: Certain oncogenes promote tumors by helping damaged cells evade programmed death.

• Tumor suppressor geneRelated: Some tumor suppressors prevent cancer by triggering apoptosis in damaged or abnormal cells.

• LymphomaRelated: Disrupted cell-death signals can let abnormal lymphocytes survive and accumulate.

• LeukemiaRelated: Disrupted cell-death signals can let abnormal leukemia cells persist.

• Retinitis pigmentosaRelated: Photoreceptor loss in retinitis pigmentosa often proceeds through programmed cell death.

• CytoplasmRelated: Its early stages include characteristic changes in cytoplasmic shape and organization.

• EcdysozoaRelated: Programmed cell death helps remodel tissues during some arthropod molts.

• Human developmentRelated: It sculpts developing tissues, including structures that change or disappear before birth.

• DNA damage responseRelated: Cells may undergo apoptosis when damage is too extensive to repair.

• FrostbiteRelated: Some cells damaged by freezing die through regulated pathways after the initial cold exposure.

• NeurotoxicityRelated: Neurotoxic exposures can trigger programmed death in neurons and glial cells.

• p53Related: When damage is severe, p53 can activate genes that initiate apoptosis.

• SyndactylyRelated: Apoptosis normally removes tissue between developing digits.

• MoultingRelated: Programmed cell death helps remove old tissues during some forms of moulting.

• ProteolysisRelated: Proteolytic cascades execute and regulate many steps of apoptosis.

• Amphibian metamorphosisRelated: Apoptosis removes larval structures such as the tadpole tail during metamorphosis.

• SunburnRelated: Heavily UV-damaged skin cells can undergo apoptosis, producing characteristic sunburn cells.

• SphingolipidRelated: Ceramide can participate in signaling pathways that promote cell death.

• Ubiquitin–proteasome systemRelated: Ubiquitin-dependent turnover can regulate proteins that promote or restrain cell death.

• Alkylating agentRelated: Unrepaired alkylation damage can activate cell-death pathways.

• Cell-cycle checkpointRelated: Severe or irreparable damage can trigger death instead of checkpoint recovery.

• Eukaryotic cellRelated: It shows how eukaryotic cells coordinate compartment breakdown during cell death.

• Sydney BrennerRelated: Worm genetics helped reveal genes that control programmed cell death.

• Leber's hereditary optic neuropathyRelated: Retinal ganglion cell loss in LHON involves programmed cell-death pathways.

• CaspaseRelated: Several caspases execute apoptosis by dismantling cellular structures.

• Somatic cellRelated: It eliminates damaged or unneeded somatic cells.

• Imaginal discRelated: During metamorphosis, apoptosis removes larval tissues while imaginal discs expand.

• RadiobiologyRelated: Severe radiation damage can activate this form of programmed cell death.

• Aaron CiechanoverRelated: Protein degradation helps regulate components that promote or inhibit apoptotic signaling.

• Benign tumorRelated: Disrupted balance between cell growth and cell death can contribute to tumor formation.

• Burkitt lymphomaRelated: Rapid proliferation makes the tumor highly dependent on survival mechanisms that restrain cell death.

• NeoplasmRelated: Neoplastic cells may evade apoptosis and persist when normal cells would die.

• Arsenic trioxideRelated: Arsenic trioxide can trigger programmed cell death in leukemia cells.