Linked from

The 23 pages that link to Bradycardia, each with the reason it gives.

• AtropineRelated: Atropine can raise heart rate when excessive vagal influence causes symptomatic bradycardia.

• HypothermiaRelated: Progressive cooling slows the heart and can make its electrical activity unstable.

• Beta blockerRelated: Excessive slowing is a direct consequence of beta blockade at the heart.

• Diving physiologyRelated: During a dive, a reduced heart rate lowers cardiac oxygen demand.

• Calcium channel blockerRelated: Verapamil and diltiazem can suppress heart rate and cardiac conduction.

• Anorexia nervosaRelated: Severe energy restriction can slow the heart and require urgent medical assessment.

• PropranololRelated: Reduced beta-1 stimulation can make propranolol slow the heart excessively.

• Deep divingRelated: Heart-rate slowing during dives helps reduce the rate at which stored oxygen is consumed.

• VerapamilRelated: Excessive slowing of nodal activity is a potential effect of verapamil.

• ClonidineRelated: Reduced sympathetic drive from clonidine can slow the heart, especially in susceptible patients.

• NeostigmineRelated: Muscarinic stimulation by increased acetylcholine can slow the heart rate.

• RhynchocephaliaRelated: Tuatara can sustain exceptionally low heart rates, consistent with their slow metabolism.

• DiltiazemRelated: Excessive sinus slowing is a possible consequence of diltiazem therapy.

• DonepezilRelated: Donepezil can increase vagal effects on the heart and raise the risk of a slow pulse.

• Neurogenic shockRelated: Unopposed vagal influence and disrupted sympathetic input can slow the heart, unlike many shock states.

• PilocarpineRelated: Systemic muscarinic stimulation can slow the heart, one concern with pilocarpine exposure.

• MetoprololRelated: Excessive slowing of heart rate is a key possible effect of beta₁ blockade.

• Earl BakkenRelated: Severe postoperative bradycardia was among the problems an external pacemaker could address.

• TimololRelated: Timolol can slow the heart by blocking cardiac beta-1 receptors.