Linked from

The 15 pages that link to Dopamine D2 receptor, each with the reason it gives.

• MetoclopramideRelated: Blocking these receptors in the chemoreceptor trigger zone reduces signals that provoke vomiting.

• Postoperative nausea and vomitingRelated: Dopamine-mediated emetic signaling explains why D2 antagonists can prevent symptoms.

• AntiemeticRelated: D2-receptor blockade explains the antiemetic action of medicines such as metoclopramide.

• AripiprazoleRelated: Aripiprazole partly activates this receptor while limiting its signaling when dopamine activity is high.

• Chemoreceptor trigger zoneRelated: D2 signaling in the trigger zone helps explain why dopamine-blocking medicines suppress some forms of nausea and vomiting.

• RisperidoneRelated: Risperidone's blockade of this receptor reduces psychotic symptoms and contributes to movement-related adverse effects.

• ChlorpromazineRelated: Blocking this receptor is central to chlorpromazine’s antipsychotic action.

• HaloperidolRelated: Haloperidol’s principal therapeutic action is antagonism at this receptor.

• OlanzapineRelated: Olanzapine’s blockade of this receptor contributes to its antipsychotic effects.

• PromethazineRelated: Promethazine's dopamine-receptor blockade contributes to its antiemetic effects.

• QuetiapineRelated: Quetiapine's relatively transient blockade of this receptor contributes to its antipsychotic action.

• FluphenazineRelated: Fluphenazine's antipsychotic action chiefly comes from blocking this receptor.