Linked from

The 18 pages that link to Fibrosis, each with the reason it gives.

• InflammationRelated: Persistent inflammation can promote scarring and fibrosis in affected tissues.

• Extracellular matrixRelated: Persistent matrix production turns ordinary repair into stiffening and scarring.

• Connective tissueRelated: Overactive matrix production turns connective-tissue repair into organ-scarring disease.

• Cellular senescenceRelated: Persistent senescent-cell signals can promote tissue remodeling and fibrotic disease.

• MechanotransductionRelated: Stiffened tissue can sustain signaling that promotes further matrix production.

• LymphedemaRelated: Chronic lymphedema can cause fibrotic tissue changes that make swelling less reversible.

• GranulomaRelated: Long-lasting granulomatous inflammation can leave fibrotic tissue behind.

• FibroblastRelated: Overactive or persistent fibroblasts drive matrix accumulation in many fibrotic disorders.

• Muscular dystrophyRelated: In dystrophic muscle, fibrosis contributes to stiffness and loss of contractile function.

• Scarring alopeciaRelated: Fibrosis replaces destroyed follicles with scar tissue.

• Autoimmune hepatitisRelated: Persistent inflammation can produce progressive liver fibrosis.

• SarcoidosisRelated: Why chronic granulomatous inflammation progresses to irreversible lung fibrosis in some people remains uncertain.

• StenosisRelated: Scar tissue can contract or thicken around a passage and reduce its opening.

• Connective tissue cellsRelated: Overactive matrix-producing cells drive the scarring that defines fibrosis.

• Musculoskeletal diseasesRelated: Fibrotic changes can stiffen tissues and restrict movement.

• Parenchymal tissue (animal organs)Related: Fibrotic expansion of stroma can replace or disrupt functional parenchyma.

• Toxic injuryRelated: Chronic toxic injury can trigger scarring that persists after exposure ends.