Linked from

The 22 pages that link to Human papillomavirus, each with the reason it gives.

• CarcinogenesisRelated: High-risk types promote carcinogenesis by disrupting cellular growth-control pathways.

• Skin cancerRelated: Certain types may contribute to squamous cell cancers, especially in immunosuppressed people.

• Cervical cancerRelated: Persistent infection with high-risk types initiates most cervical cancers.

• p53Related: High-risk HPV E6 protein promotes p53 degradation in infected cells.

• Oral sexRelated: Oral exposure to HPV can cause infection and is linked to some throat cancers.

• CervixRelated: Persistent infection with high-risk types causes most cervical cancers.

• DysplasiaRelated: High-risk types drive many cervical dysplastic lesions and some other epithelial abnormalities.

• Henrietta LacksRelated: Research on cervical cancer cells, including HeLa, contributed to understanding the role of viruses in cancer.

• Anal cancerRelated: Persistent high-risk HPV infection drives most anal squamous cell cancers.

• Dental damRelated: HPV can spread through skin contact beyond the area a dental dam covers.

• Penile cancerRelated: Persistent high-risk HPV infection contributes to a subset of penile tumors.

• FellatioRelated: Certain types can infect the mouth or throat after sexual transmission.

• Head and neck cancerRelated: HPV-associated oropharyngeal tumors differ biologically and often have a better prognosis.

• Irwin RoseRelated: The viral E6 protein exploits ubiquitin-mediated degradation to eliminate the tumor suppressor p53.