Linked from

The 12 pages that link to Molecular mimicry, each with the reason it gives.

• AutoimmunityRelated: A response to infection can mistakenly recognize similar host structures.

• Guillain–Barré syndromeRelated: Antibodies raised against some infections can cross-react with molecules on peripheral nerves.

• Molecular recognitionRelated: Mimics exploit recognition by presenting features similar to those of a natural binding partner.

• Autoimmune diseaseRelated: A response to an infection can sometimes cross-react with a similar self-antigen.

• Streptococcus pyogenesNarrower topic: It helps explain how antibodies generated against streptococcal antigens may also target human tissues.

• Host–parasite coevolutionRelated: Parasites can evolve host-like molecules to evade recognition, prompting further host defenses.

• Rheumatic feverRelated: Similarity between streptococcal and human molecules helps explain why infection can trigger inflammation in the heart and other tissues.

• Reactive arthritisRelated: Shared microbial and human features may help explain how infection triggers inflammation at distant joints.

• Group A StreptococcusRelated: It helps explain how an immune response to streptococcal antigens can contribute to rheumatic fever.

• Capsular polysaccharideRelated: Some capsules resemble host carbohydrates and thereby evade immune attack.

• Campylobacter jejuniRelated: Structural resemblance between C. jejuni surface sugars and nerve gangliosides can misdirect immunity.

• Immune System DiseasesRelated: Cross-reactivity can connect an infection to autoimmune tissue damage.