Linked from

The 14 pages that link to NMDA receptor, each with the reason it gives.

• Nitrous oxideRelated: Nitrous oxide inhibits these receptors, contributing to analgesia and anesthesia.

• GlutamateRelated: NMDA receptors turn glutamate signaling into calcium entry and synaptic plasticity.

• GABA receptorCompared with: It contrasts with GABA-A receptors as a major excitatory channel with distinct plasticity roles.

• Synaptic plasticityRelated: Its calcium entry can trigger molecular pathways that change synaptic strength.

• KetamineRelated: Ketamine blocks this receptor, a central part of its anesthetic and dissociative action.

• Long-term potentiationRelated: Its coincidence detection helps initiate many forms of potentiation.

• SevofluraneRelated: Sevoflurane also suppresses excitatory NMDA-receptor signaling.

• DextromethorphanRelated: At high concentrations, dextromethorphan and its metabolite inhibit this receptor.

• IsofluraneRelated: Isoflurane can reduce excitatory signaling involving NMDA receptors.

• AnestheticRelated: Some anesthetics reduce central excitation by inhibiting this receptor.

• PhencyclidineRelated: PCP binds within this receptor’s channel and inhibits its activity.

• Anti-NMDA receptor encephalitisBroader topic: The autoantibodies target this receptor and reduce its availability at neuronal synapses.

• General anaestheticRelated: Ketamine produces anaesthesia partly by blocking this receptor.

• IbogaineRelated: Ibogaine interacts with this receptor, one of several proposed contributors to its effects.