Linked from

The 25 pages that link to Tumor microenvironment, each with the reason it gives.

• Extracellular matrixRelated: Tumor matrix can alter cancer-cell behavior, immune access, and treatment response.

• AdenosineRelated: High extracellular adenosine can suppress antitumor immune activity in this setting.

• MetastasisNarrower topic: Local stromal and immune signals can shape invasion and metastatic potential.

• Colorectal cancerRelated: Immune cells and stromal signals help determine how colorectal tumors grow and respond to therapy.

• Lung cancerRelated: Immune and stromal cells around lung tumors can influence growth and treatment response.

• MelanomaRelated: Immune and stromal cells around melanoma can either restrain or support its growth.

• ImmunotherapyRelated: Its suppressive features can limit treatment effectiveness and shape resistance.

• LymphomaRelated: Noncancerous immune and stromal cells can support or restrain lymphoma growth.

• Lymph nodeNarrower topic: Tumor-draining nodes are part of the wider environment shaping antitumor immunity.

• Epithelial–mesenchymal transitionRelated: Signals from this environment can induce or shape EMT-like states in cancer cells.

• Hepatocellular carcinomaRelated: Liver immune cells and scar tissue influence tumor growth and response to therapy.

• Cancer immunotherapyRelated: Its suppressive signals can prevent immune cells from attacking tumors effectively.

• Paracrine signalingRelated: Paracrine exchanges between tumor and surrounding cells can support growth, invasion, or immune escape.

• Antibody-dependent cellular cytotoxicityNarrower topic: Its suppressive signals and limited immune access can constrain ADCC in solid tumors.

• OncologyRelated: Its immune and structural components can affect tumor growth and treatment response.

• Cancer researchRelated: It explains why cancer behavior depends on surrounding tissue as well as tumor cells.

• Hodgkin lymphomaRelated: Most of a Hodgkin lymphoma mass can consist of recruited immune cells rather than tumor cells.

• Pancreatic cancerNarrower topic: Pancreatic tumors depend on interactions between cancer cells, immune cells, and dense surrounding tissue.

• Liver cancerRelated: Its immune and structural features influence liver tumor growth and treatment response.

• Reed–Sternberg cellsRelated: The abundant immune cells around Reed–Sternberg cells help sustain the lymphoma.

• SarcomaRelated: Blood vessels, immune cells, and connective tissue can shape sarcoma growth and treatment response.

• PhosphatidylserineRelated: Tumor cells can display phosphatidylserine, shaping local immune and vascular interactions.

• DurvalumabRelated: PD-L1 expression within this environment can suppress immune attack and shape treatment response.

• EmperipolesisRelated: Cell-in-cell interactions in tumors raise questions about emperipolesis-like events and their effects on both cells.

• Pancreatic stellate cellsNarrower topic: Activated stellate cells are stromal components that shape this environment in pancreatic cancer.