Linked from

The 75 pages that link to Inflammation, each with the reason it gives.

• Wound healingRelated: Early inflammation clears microbes and damaged material while signaling cells involved in repair.

• AtherosclerosisRelated: Persistent inflammation helps sustain plaque growth and can weaken its protective fibrous cap.

• Nonsteroidal anti-inflammatory drugNarrower topic: NSAIDs suppress parts of this response rather than removing its underlying cause.

• CytokineNarrower topic: Cytokines coordinate many of the signals that initiate and sustain inflammation.

• PhagocytosisRelated: Phagocyte activity can initiate, amplify, or resolve inflammatory responses.

• CortisolRelated: Cortisol limits inflammatory signaling, which underlies some therapeutic uses and risks.

• Acne vulgarisRelated: Inflammation turns follicular blockage into red, tender acne lesions.

• Immune systemRelated: It brings immune cells and molecules to threatened or damaged tissue.

• EdemaCompared with: Inflammation may produce edema, but it is a broader biological response rather than fluid accumulation alone.

• CellulitisNarrower topic: Inflammation produces the redness, warmth, swelling, and pain seen in infected skin.

• Omega-3 fatty acidRelated: Omega-3-derived lipids can influence inflammatory signaling and its resolution.

• CyclooxygenaseRelated: Inflammatory signals can increase COX-2 expression and prostaglandin production.

• InfectionCompared with: Inflammation may result from infection, but can also arise without one.

• ProstaglandinNarrower topic: Prostaglandins help shape the vascular and sensory components of inflammatory responses.

• PneumoniaNarrower topic: Inflammation drives swelling and fluid buildup in infected lung tissue.

• Innate immune systemRelated: Innate detection often triggers inflammation to recruit defenses and contain damage.

• AnalgesicRelated: Inflammation produces mediators that contribute to pain and are targets of NSAIDs.

• Arachidonic acidRelated: Several arachidonic-acid metabolites promote or resolve inflammatory responses.

• AntihistamineNarrower topic: Histamine contributes to inflammation, though antihistamines do not suppress every inflammatory pathway.

• PathologyRelated: It is a common response that shapes tissue damage and repair.

• EicosanoidNarrower topic: Eicosanoids help coordinate inflammatory onset, intensity, and resolution.

• DermatologyNarrower topic: Inflammatory pathways underlie many rashes and shape their signs and symptoms.

• Adaptive immune systemRelated: Innate inflammation helps initiate and shape adaptive responses.

• Interleukin 6Narrower topic: IL-6 helps coordinate both local inflammation and its systemic effects.

• C-reactive proteinNarrower topic: CRP concentration reflects inflammatory signaling rather than a single underlying disease.

• DiseaseRelated: Inflammation can be a defense, a disease mechanism, or both.

• NecrosisRelated: Necrotic cell contents can signal damage and initiate this response.

• Paracrine signalingRelated: Paracrine cytokines and other mediators coordinate responses among nearby cells at affected tissue.

• Tumor necrosis factorNarrower topic: TNF is one of the signals that initiates and sustains this broader response.

• TurmericRelated: Anti-inflammatory effects are a major proposed explanation for curcumin’s studied health benefits.

• BiocompatibilityRelated: Inflammation is a common response used to judge how tissue reacts to materials.

• Spider biteRelated: Local inflammation explains many ordinary symptoms around a bite.

• Wound careRelated: Some inflammation is part of repair, but excessive or persistent inflammation can delay healing.

• Acute painRelated: Inflammation releases mediators that can produce tenderness and sensitize pain pathways.

• Alpha-linolenic acidRelated: Omega-3 metabolism can alter lipid signaling pathways associated with inflammatory responses.

• Linoleic acidRelated: Research examines whether linoleic-acid metabolism alters inflammatory signaling, rather than treating the fatty acid as inflammation itself.

• BradykininRelated: Bradykinin is one mediator that helps produce vascular changes and pain during inflammation.

• Bacterial infectionRelated: Inflammation produces many signs and symptoms during bacterial disease.

• Élie MetchnikoffRelated: Metchnikoff connected the arrival of phagocytes at injured tissue with cellular defense.

• Omega-6 fatty acidRelated: Omega-6-derived mediators participate in inflammatory signaling, but effects vary by molecule and context.

• TendinopathyCompared with: Tendinopathy is not synonymous with inflammation, despite inflammatory signaling sometimes occurring in tendon tissue.

• FistulaRelated: Persistent inflammation can damage tissue and sustain a fistula.

• PharyngitisNarrower topic: Its local immune response accounts for much of the soreness and swelling.

• SalmonellosisRelated: The intestinal inflammatory response contributes to diarrhea and other symptoms.

• Tinea corporisRelated: Local inflammation makes the advancing edge red, raised, and scaly.

• AcneNarrower topic: Inflammatory responses produce the redness, swelling, and tenderness of many acne lesions.

• ArthritisNarrower topic: Inflammation drives pain and swelling in many arthritides, though not all joint damage is inflammatory.

• CaspaseRelated: Inflammatory caspases process cytokines that help initiate immune responses.

• RheumatologyRelated: Persistent inflammation drives pain and tissue damage in many conditions rheumatologists treat.

• Abdominal painNarrower topic: Inflammation is a common mechanism behind painful abdominal disorders.