Linked from

The 13 pages that link to Ischemia-reperfusion injury, each with the reason it gives.

• ApoptosisRelated: Oxidative and metabolic stress during reperfusion can contribute to apoptotic cell loss.

• Organ transplantationRelated: An organ can be injured during preservation and again when circulation is restored.

• Reactive oxygen speciesRelated: Reoxygenation can trigger a surge in reactive oxygen species and cellular injury.

• Oxidative stressRelated: Reoxygenation can produce a burst of reactive molecules that worsens tissue injury.

• HibernationRelated: Hibernators tolerate physiological stresses that would injure many nonhibernating mammals.

• HypoxiaRelated: Restoring oxygen after deprivation can itself provoke oxidative and inflammatory injury.

• AutophagyRelated: Autophagy can shape cellular responses to the stress of oxygen loss and restoration.

• NeutrophilRelated: Recruited neutrophils can intensify injury through oxidants and inflammatory enzymes.

• IschemiaRelated: Restored flow can trigger oxidative and inflammatory injury in previously ischemic tissue.

• Cold storage of organsRelated: Cold storage limits injury during ischemia, but reperfusion after implantation can trigger a second wave of damage.

• Programmed cell deathRelated: Reperfusion can trigger several regulated death pathways in oxygen-deprived tissue.

• Cell deathRelated: Restored oxygen and inflammation can intensify cell death after ischemia.

• HypoxanthineRelated: ATP breakdown during ischemia can raise hypoxanthine, which is oxidized during reperfusion.