Linked from

The 49 pages that link to Multiple sclerosis, each with the reason it gives.

• Blood–brain barrierRelated: Inflammation can increase barrier permeability and enable immune-cell entry into the brain.

• NeuronRelated: Its effects show how loss of axonal insulation impairs neuronal communication.

• Cerebrospinal fluidRelated: Distinctive immune proteins in cerebrospinal fluid can support its diagnosis.

• Central nervous systemRelated: It shows how damage to myelinated pathways can impair movement, sensation, and other functions.

• MyelinRelated: Inflammatory injury to myelin is a defining feature of this disease.

• AxonRelated: Damage to myelin disrupts signaling along affected axons.

• Lumbar punctureRelated: Fluid analysis may show oligoclonal bands that support diagnosis in appropriate cases.

• OligodendrocyteRelated: Oligodendrocyte and myelin loss disrupts signaling and contributes to neurological disability.

• White matterRelated: Demyelinating lesions can damage white-matter pathways and impair communication between regions.

• DysarthriaRelated: Lesions affecting speech motor pathways can produce varied dysarthria patterns.

• NeuroanatomyRelated: Its lesions illustrate how damage to white-matter pathways disrupts neural function.

• Epstein–Barr virusRelated: Strong epidemiologic evidence links prior EBV infection to multiple sclerosis, but the causal pathway remains under study.

• Molecular mimicryRelated: Pathogen–myelin resemblance has been investigated as one possible contributor to disease onset.

• Saltatory conductionRelated: Demyelination can disrupt saltatory conduction and slow or block impulses.

• SpasticityRelated: Spinal cord and brain lesions can cause stiffness, spasms, and spasticity.

• Urinary incontinenceRelated: Nerve disruption can interfere with bladder signaling and control.

• SphingolipidRelated: Myelin’s sphingolipid-rich structure is disrupted during the disease.

• Optic neuritisRelated: Optic neuritis can be the first clinical sign of multiple sclerosis.

• Cerebellar ataxiaRelated: Lesions in cerebellar pathways can produce ataxia among varied neurological symptoms.

• InterferonRelated: Interferon beta is used to reduce relapses in some forms of this disease.

• Theodor SchwannRelated: Its effects highlight the contrast between peripheral Schwann-cell myelin and central nervous system myelin.

• Corpus callosumRelated: Lesions can affect callosal fibers and appear as characteristic abnormalities on brain MRI.

• Peter MedawarRelated: Medawar developed multiple sclerosis later in life and wrote about its effects on his work.

• Jacqueline du PréRelated: The disease ended du Pré’s cello career as her coordination and physical control declined.

• GliaRelated: The disease illustrates the consequences of myelin damage and oligodendrocyte loss.

• HemiplegiaRelated: A lesion affecting motor pathways can cause hemiplegia or severe one-sided weakness.

• Michael RedgraveRelated: Redgrave lived with multiple sclerosis, which affected his health during his later years.

• Blood–spinal cord barrierRelated: Barrier changes can accompany inflammatory lesions in the spinal cord.

• HypoesthesiaRelated: Lesions can interrupt sensory pathways and cause patchy sensory loss.