Linked from

The 17 pages that link to Nephrotoxicity, each with the reason it gives.

• CisplatinRelated: Kidney damage is a major dose-limiting toxicity of cisplatin and motivates hydration protocols.

• Mercury poisoningRelated: Inorganic mercury can accumulate in and damage kidney tissue.

• AminoglycosideRelated: Renal accumulation can injure proximal tubule cells during aminoglycoside treatment.

• Renal impairmentRelated: Potentially kidney-toxic treatments can worsen existing impairment.

• ChloroformRelated: Chloroform exposure can also harm the kidneys, especially at substantial doses.

• StreptomycinRelated: Renal toxicity is a serious risk during streptomycin treatment.

• Amphotericin BRelated: Amphotericin B can reduce kidney blood flow and damage tubular cells.

• TacrolimusRelated: Tacrolimus can impair kidney function, complicating care after transplantation.

• DimercaprolRelated: Metal–dimercaprol complexes can be harmful to the kidneys, especially when renal function is impaired.

• NeomycinRelated: Systemic neomycin exposure can damage kidney tubules, limiting its use beyond topical treatment.

• Carbon tetrachlorideRelated: CCl₄ exposure can also injure kidney tissue, particularly after substantial doses.

• AmikacinRelated: Amikacin can accumulate in renal tubules and impair kidney function during treatment.

• Metal toxicityRelated: The kidneys accumulate and excrete many metals, making them common targets of toxicity.

• TobramycinRelated: Kidneys clear tobramycin, and accumulation can injure renal tubules.

• CiclosporinRelated: Kidney toxicity is a major dose-limiting risk requiring monitoring during treatment.

• Sodium calcium edetateRelated: Edetate is cleared through the kidneys and can cause renal injury, especially with excessive dosing.